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1.Higher rates of bacterial vaginosis（BV）among black women likely contribute to disparity in these outcomes.Unfortunately, the primary causes of disparity in BV prevalence are not known.2.This has led to speculation that black women are predisposed to BV because of inherently higher pH levels.3.Racial differences in vaginal pH may simply reflect differences in vaginal flora.4.Comparisons of pH levels by race that do not account for differences in flora may yield spurious results.5.To addre this iue, we compared vaginal pH among a racially diverse sample of pregnant women without BV and trichomoniasis, and stratified comparisons by vaginal floral patterns.6.Details regarding the study have been previously described.7.The current analysis of these previously collected data was exempted from the Institutional Review Boards(IRBs)review by the Office of Human Subject Research, National Institutes of Health.8.Race/ethnicity was based on 5 mutually(5对）exclusive categories: white, black,Hispanic, Asian-Pacific Islander, or other
9.Sterile cotton swabs were used to obtain material from the posterior vaginal fornix for a vaginal smear and for culture for Trichomonas vaginalis(T.vaginalis)
10.Cultures were incubated at 37(C;at 2-to 3-day intervals, samples were examined microscopically under 400!power for the presence of motile, oval flagellated protozoa.11.Vaginal smears were air dried, Gram stained, and aeed under magnification(x1000)using the scoring system of Nugent et al.12.This finding was confirmed in a multivariate analyses that included race,actual score value, age and clinical site.13.The adjusted difference in vaginal pH was no longer statistically significant(0.015 lower in white women, 95% Confidence Interval;-0.036-0.007, p=0.18).14.Given these findings, it is likely that racial variation in vaginal pH is attributable to differences in vaginal flora and not to biological variation in vaginal pH.15.Experimental data are consistent with the hypothesis that lactobacilli maintain vaginal pH in the normal range presumably by the anaerobic metabolism of vaginal glycogen to acidic products.16.Therapeutic use of carbohydrates in inflammatory bowel diseases(IBDs)is discued from two theoretical, apparent diametrically opposite perspectives: regular ingestion of prebiotics（益生元）or withdrawal of virtually all carbohydrate components.17.Pathogenesis of IBD is discued connecting microbial flora, host immunity, and genetic interactions.18.The conclusion reached is that, while both approaches may alleviate symptoms in both IBS and IBD, there is insufficient data yet to determine whether both approaches lead to equivalent bacterial effects in mollifying the immune system.19.In particular, Crohn’s disease(CD)and idiopathic ulcerative colitis(UC)(the two clinical subtypes of IBD)were targeted to capitalize on the potential therapeutic effects of either pro-or prebiotics.20.
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Beginningl In this paper, we focus on the need for l This paper proceeds as follow.l The structure of the paper is as follows.l In this paper, we shall first br......
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浅谈高中英语写作中语域知识的习得总论：新课程标准中＂语域＂的概念在中学阶段英语教学中是个新鲜词汇，第一次提到，并且明确列入评分标准之中。语域知识的习得是学生写作能力发展过......
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